Melanotan II Posted on 10 Apr 20:57 , 0 comments
By Todd Lee M.D.
Melanotan II
What if I told you there was a drug that prevented skin cancer, prevented diabetes, cured impotence and made you look better? You would say, if a drug was this great you would have heard of it. Which might be true, if not for the FDA. For almost a decade, Europeans have been benefiting from the miracle drug Melanotan II. In fact, in 2009, a poll of Norwegian pharmacies deduced that 10,000 syringes were used by melanotan users. That's one country and one year.
Want to know the biggest irony? It was developed in Arizona in 1996. It was developed to prevent skin cancer because it causes the user to develop a tan. Because of this, Britain and Norway are favorites of it. You see, the FDA won't legalize it because it was being researched for release as a separate drug, one that causes erections.
Let me clarify this: a drug was developed in the United States for prevention of skin cancer that protects you from sunlight by giving you a tan but the side effect was it gave you erections. Because of the greed of corporate America, since there is more money in erections than preventing cancer, the drug has been tied up in testing for 18 years. Meanwhile, all of Europe has been using it for over a decade. What's the FDA’s excuse? They claim it causes skin cancer. WTF you ask? Let me explain the science.
Put Your Depends On, This Is Gonna Be Rough!
Sunlight has ultraviolet beams. 3 types penetrate our O-zone. A, B, and C. Sunlight has lots of A which do the least damage and create a superficial tan which is short lived and doesn't cause melanocytes to release more melanin, it only darkens existing melanin. Type B rays do way more DNA damage and subsequently, the body makes permanent changes to protect against the damage. The melanocytes release more melanin to protect the body. This allows the body to get darker faster when exposed to UVA beams. C and the high energy beams ionize atmospheric nitrogen and reflect off the ozone respectively. Clouds filter the light and protect people from the sun. Arizona is sunny, Norway is not. So with more sunlight hitting the skin of people, you get more skin cancer. There is damage to the DNA. UVA does single strand breakage and UVB does double strand breakage and causes non functional bonds. If the DNA repair does not happen right, skin cancer may result. Eventually, given enough UV radiation all people will get skin cancer.
Genetics of the repair mechanism is half the reason why some people get skin cancer and others don’t. The other half of the genetics is their natural propensity for skin pigmentation. Melanocytes are cells in the skin that release melanin. Melanin is what gives pigment. Brown hair, brown eyes and brown skin all come from melanin. Fair skin, blonde hair and blue eyes are due to double recessive genes for low melanin. So in prehistoric times, people who had sunny climate, like Africa, evolved to have melanin: the dark pigmentation protected their skin from the UV damage. In places cloudy and cold like Norway, my ancestors developed light pigmentation to allow as much UV light as possible in, since that UV energy was necessary to form Vitamin D. 20 minutes for fair people of direct sunlight can generate as much as 1000 IU of Vitamin D.
How is this relevant? Well, when you take people whose ancestors come from Norway and put them in a sunny desert like Arizona, their skin doesn't have the melanin armor to protect it from the sun. Subsequently fair people are the ones most susceptible to skin cancer: their skin takes so much DNA damage they can't repair enough of it and it’s a matter of time…
Tanning is a way of exposing yourself to controlled amounts of UV light to change the skin color. After exposure to damaging UVB rays, the melanocytes release more melanin to protect the body from more sunlight damage. It's a risky proposition though, to expose yourself to sunlight, to protect yourself from sunlight.
Alpha Melanocyte stimulating hormone binds to receptors all over your body. One of these receptors is on the melanocyte. This bonding of hormone and receptor stimulates melanin release. University of Arizona researchers correctly hypothesized that injecting A-MSH would cause the melanocytes to release melanin but the hormone had a short half life. They modified it to have a longer half life and to stimulate the melanocyte 1000 times more effectively!
This they christened “Melanotan.” When tested on men, they found that 5 hours after administration, erections were a side effect.
Now, 18 years later, it still isn't legal. The erection side effect has resulted in the big money pharmaceutical companies use as influence over our corrupt government to sequester the life saving technology so they can have the next billion dollar industry changer like Viagra or Prilosec. Pharmaceutical company accountants sacrifice thousands of lives to get a bigger paycheck. Only in America. No literally, only in America. In the civilized countries of Europe, where politicians are less corrupt, this drug is used widespread. Like I said, 10,000 doses were used in 2009 in Norway alone. One country, one year. It also up regulates GLUT-4 transporter activity, so it shuttles glucose into skeletal muscle. This would improve insulin sensitivity and combat diabetes, aid muscle building and aid fat loss.
Do you know someone who was fair and died of skin cancer in the last 18 years? Do you miss them? Thank the FDA. Their argument is that melanocyte stimulation can cause melanoma. That dark moles may become irregular. One has to have moles and freckles for this to happen though. So not all people are going to get dark spots but all people will be protected from sunlight. So to protect some, they condemn all. Faulty logic at best.
A woman I know had 7 pieces of her skin removed because her dermatologist was convinced she had skin cancer. She explained that she used melanotan and he admitted HE HAD NO IDEA WHAT IT WAS. She tried to educate him but he wouldn’t listen, he is a doctor after all, we know everything, lol. So, 7 patches of skin and a $1700 bill later she took me to the appointment to see if it was in fact cancer. “Benign Nevus” was the diagnosis I had to force out of him. He couldn't make eye contact. “So they were moles, not cancer. Like she told you.” No response. “So the melanotan did exactly what it's supposed to and made the melanin darker so the moles got darker at a faster rate. Like she tried to explain.” No answer. This “Healer” Dr. Lipkin of Brighton Michigan is still cutting people up, probably as we speak. Everyone I have ever met who has went to him, he has cut some of their skin off and in only one case, it might have been cancer. That person had a family history of skin cancer and tanned extensively. Had she used Melanotan instead… he would likely cut her anyway but it would have been benign.
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